Just a quick inspection of the hair makes me suspicious of Mr. Lanza. Don't need no genealogy.
but you don't believe in science 87...what gives?
likely just an attempt to get money b/c I think the biggest deciding factors on an individual's behavior whether it be good or evil actions are nature/nurture
Here is an abstract where they are finding mutations that may cause schizophrenia
Considering the key role of d-serine in N-methyl-d-aspartate receptor-mediated neurotransmission, it is highly relevant to define the role that enzymes play in d-serine synthesis and degradation. In particular, the details of regulation of the d-serine catabolic human enzyme d-amino acid oxidase (hDAAO) are unknown although different lines of evidence have shown it to be involved in schizophrenia susceptibility. Here we investigated the effect of three single nucleotide polymorphisms and known mutations in hDAAO, i.e., D31H, R279A, and G331V. A very low amount of soluble G331V hDAAO is produced in E. coli cells: the recombinant variant enzyme is fully active. Human U87 glioblastoma cells transiently transfected for G331V hDAAO show a low viability, a significant amount of protein aggregates, and augmented apoptosis. The recombinant D31H and R279A hDAAO variants do not show alterations in tertiary and quaternary structures, thermal stability, binding affinity for inhibitors, and the modulator pLG72, whereas the kinetic efficiency and the affinity for d-serine and for FAD were higher than for the wild-type enzyme. While these effects for the substitution at position 31 cannot be structurally explained, the R279A mutation might affect the hDAAO FAD-binding affinity by altering the "structurally ambivalent" peptide V47-L51. In agreement with the observed increased activity, expression of D31H and R279A hDAAO variants in U87 cells produces a higher decrease in cellular d/(d+l) serine ratio than the wild-type counterpart. In vivo, these substitutions could affect cellular d-serine concentration and its release at synapsis and thus might be relevant for schizophrenia susceptibility.
Copyright © 2012. Published by Elsevier B.V.
But yeah, leave it to 87 to simplify science into calling it quackery.
This is what you said:
but attempting to tie one gene to evil behavior is not science....it's quackery....
2) The first step towards solving a common problem amongst several different subjects is to find a common cause. Which is exactly what scientists trying to do with evidence and fact based techniques. I know a difficult concept to grasp for you since the paper didnt simply say "God said it so you know, just believe me and have faith. And dont forget to run with it while with clenched fists calling everyone else that doesnt run with you as sinners"
and you dishonestly translated what i said....
and yes.....you put viruses into the same scientific context as human behavior...
Just a continuation of a long standing debate between cultural anthropology and cognitive scientists/evolutionary psychology. Anthropologists insist there are actually very few genetically driven behaviors (fear of falling, sex drive, suckling, etc.), but of cognitive scientists, lead most notably by Steven Pinker, are bringing forth serious challenges to those long held beliefs.
Ashley Montagu would have been one of the older and well known people in the anthropology camp to hold the view than human agression is entirely learned behavior.